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82 Additionally, Aβ 1–42 M35L substitution in mice has also been observed to deposit similar amounts of Aβ 1–42 as well as Aβ 1–40 in punctuate forms, supporting the theses of this review that oxidation of M35 residue of Aβ 1–42 is implicated for plaque formation

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Folate deficiency leads to the accumulation of homocysteine which is associated with an increased risk of cerebrovascular diseases, depression, and dementia, with a focus on vascular dementia and Alzheimer’s disease (4, 20, 22)