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While COX-2 selective inhibitors and diclofenac are associated with greater cardiovascular but lower gastrointestinal risks than non-selective NSAIDs, non-selective NSAIDs demonstrate the opposite effect
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Among the identified compounds, 1-(3,4-dihydroxyphenyl)-6,7-dihydroxy-1,2-dihydro-2,3-naphthalenedicarboxylic acid, taxifolin, and 1- O -caffeoyl-6- O -( S )-brevifolincarboxyl-β-D-glucopyranose were predicted to be the principal contributors to this superior inhibitory effect
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